The blog is about health and gives useful information on health and disease.

Gautam had just finished his University Exams. The results looked far away. He gave himself up to reading different books by various authors God knows whether it was the influence of some reading or something else, but Gautam suddenly seemed to have lost interest in everything – no more reading, no more visits to friends or outings, no interest in eating – Most of the time he lay dosing or sleeping and the few hours that he was awake, he sat alone on a bench in a park looking away doing nothing, only musing. His parents were worried. The clinical tests showed ‘All clear’.

For us it was a clear case of Clematis negative state and Clematis remedy brought Gautam once more ‘to earth’. Inattentiveness of Clematis is different from that of Chestnut Bud.

CLEMATIS patient has always a pre-occupied mind -a mind full of fantasis, dreams of rosy future, and occupied with building castles in the air. There is, in effect, no place in his mind to think of the present.

CHEST- NUTBUD patient lives very much in the present. He does not have a pre-occupied mind. Only he does not pay attention to things in which he is not interested, and therefore does not observe properly and makes the same mistakes again and again.

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Until very recently, the party line was that from the perspective of bone density, Caucasians and Asians get the short end of the stick compared to African-American, Hispanic and Polynesian people. I was taught that thanks to gene pool differences, and a greater tendency to have small frames, white and Asian people have a significantly higher risk of losing too much bone mass, developing arthritis and suffering fractures. But I recently learned from Dr. Juanita Archer, a metabolic bone specialist at Howard University, that the most recent studies indicate that African-Americans have the same risk as Caucasians. Historically, black people were presumed not to be much at risk for osteoporosis and so by and large weren’t screened for it until there was already a fracture. When doctors challenged the assumptions they’d been working under, and started looking for low bone density in black women at random, they found it was prevalent in them, too. We don’t have the data yet, but I’m guessing that when we begin to look at other ethnic groups, we’ll discover the same risks.

FAMILY HISTORY

Your grandmother probably gave you many wonderful things both tangible and intangible, but if she had osteoporosis, good bones isn’t one of them. One of the primary warnings you will have that you are at higher risk of low bone density is a history of osteoporosis in your family. If your mother, father, sibling, or grandparent suffered from it (or lost height; or had a dowager’s hump or a hunchback; or got frequent fractures or fractures from minor trauma), you’ll need to pay particular attention to your own plan to avoid a similar fate.

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Various physiological changes occur in response to the increased demand of energy, required during exercise. These include:

i) Enhanced cardiac output to meet the increased oxygen demand of the organs.

ii) Augmented respiration to help higher oxygen (02) inflow and CO? elimination from the lungs.

iii) Redistribution of blood flow and raised capillary perfusion pressure.

METABOLIC FUEL

Major metabolic fuel used by muscles are:

i) Glucose & Free fatty acids: Which are released into circulation by liver and adipose tissue.

ii) Aminoacids: These are not important but contribute less than 10% of total energy supply.

iii) Ketorfe bodies: These can be used only during low glucose availability.

METABOLIC CHANGES DURING EXERCISE

Energy utilization during exercise is mediated and influenced by Insulin and Counter regulating hormones.

A decrease in plasma insulin and presence of glucagon appears to be necessary for early increase in the hepatic productions of glucose during exercise, and during prolonged exercise, increase in plasma glucagon and catecholamines appear to play a key role.

METABOLIC FUEL SOURCES:

Intracellular Fuel Sources : Fat & Carbohydrate present in the muscles

Extracellular Fuel Source : Site – Liver -Glycogenolysis -Glucose

Lipolysis : Site – Adipose Tissue Fat – Free Fatty Acid

Neoglucogenesis : Site – Liver – Glucose

ENERGY SOURCES IN RELATION TO STATE OF EXERCISE

ACTIVITIES

ENERGYSOURCE

1.

Resting

- 90% comes for oxidation of FFA

2.

Usual work

- Oxidation of glucose & FFA

3.

During short burst of exercise

- Muscle glycogenosis followed by

hepatic glycogenolysis.

4.

If exercise continued

- Hepatic-gluconeogenesis.

5.

If exercise is beyond 30 Minutes

- ERA. generated by adipose tissue

lipolysis.

6.

Post-exercise phase

- Restoration of glycogen in muscle

& liver, which is insulin dependent.

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